Point of Care Significance 7/10

Checkpoint Inhibitor Exposure Before Allogeneic Transplant Improves PFS in Hodgkin Lymphoma

A joint CIBMTR/EBMT analysis of 2,186 adult Hodgkin lymphoma patients showed that prior checkpoint inhibitor exposure before allogeneic HCT reduced relapse incidence (HR 0.58) and improved progression-free survival, though acute GVHD rates increased. Post-transplant cyclophosphamide-based prophylaxis mitigated GVHD risk in CPI-exposed patients. These findings inform MRD monitoring strategies and flow cytometric GVHD surveillance protocols in the post-immunotherapy transplant setting.

The original study

Outcomes of allogeneic HCT in Hodgkin lymphoma in the era of checkpoint inhibitors: a joint CIBMTR and EBMT analysis.

Authors
Perales MA, Awan FT, Boumendil A, Patel J, Castagna L, Angelucci E, et al.
Journal
Blood
Type
Journal Article, Multicenter Study
PMID
40623049
Read the original study →

Original abstract

Checkpoint inhibitors (CPIs) have shown remarkable efficacy in Hodgkin lymphoma (HL), and are now used routinely. While allogeneic hematopoietic cell transplantation (allo-HCT) remains a curative option for HL, there are concerns prior CPIs may exacerbate post-allo-HCT complications, particularly graft-versus-host disease (GVHD), and lead to worse outcomes. Given the relative paucity of data, we performed a Center for International Blood and Marrow Transplant Research/European Society for Blood and Marrow Transplantation study to examine the impact of prior CPIs in allo-HCT. We included 2186 adult patients aged >18 years who received a first allo-HCT using a matched related, unrelated, or haploidentical donor from 2008 to 2023. Twenty-seven percent of patients received prior CPIs. GVHD prophylaxis was posttransplant cyclophosphamide (PTCy) in 55.8% of patients in the CPI cohort, and 35% in the non-CPI cohort. Median follow-up among survivors was longer for the non-CPI (39 months) than CPI cohort (16.5 months). In multivariate analysis, prior CPI exposure did not affect overall survival (OS) or nonrelapse mortality, but resulted in improved progression-free survival (non-CPI vs CPI hazard ratio [HR], 0.81; 0.67-0.98; P = .03) and lower relapse incidence (HR, 0.58; 0.45-0.76; P < 001). While grade 2 to 4 (HR, 1.26; 1.04-1.53; P = .02) and 3 to 4 (HR, 1.41; 1.04-1.92; P = .03) acute GVHD (aGVHD) were increased, differences in chronic GVHD (cGVHD) were not significant. PTCy-based GVHD prophylaxis resulted in improved OS, lower grade 2 to 4 aGVHD, and cGVHD in patients with prior CPI exposure. In summary, allo-HCT should still be considered a curative option for patients with HL in the era of CPIs.