Liquid Biopsy Landmark-class

ctDNA Outperforms Flow Cytometry for Early Relapse Detection in CLL Triple-Combination Therapy

Final analysis of the CLL2-BAAG trial showed that MRD-guided acalabrutinib, venetoclax, and obinutuzumab achieved undetectable MRD in 93.3% of 45 relapsed/refractory CLL patients, including those with prior BTK inhibitor exposure and TP53 aberrations. Among 18 MRD recurrences detected post-treatment, ctDNA identified 12 relapses before flow cytometry, while only 3 were detected first by flow cytometry. These findings suggest ctDNA-based monitoring meaningfully complements conventional flow cytometry for earlier detection of disease recurrence.

The original study

Acalabrutinib, venetoclax, and obinutuzumab in relapsed/refractory CLL: final efficacy and ctDNA analysis of the CLL2-BAAG trial.

Authors
Fürstenau M, Giza A, Weiss J, Kleinert F, Robrecht S, Franzen F, et al.
Journal
Blood
Type
Journal Article, Clinical Trial, Phase II, Multicenter Study, Research Support, Non-U.S. Gov't
PMID
38620072
Read the original study →

Original abstract

The phase 2 CLL2-BAAG trial tested the measurable residual disease (MRD)-guided triple combination of acalabrutinib, venetoclax, and obinutuzumab after optional bendamustine debulking in 45 patients with relapsed/refractory chronic lymphocytic leukemia (CLL). MRD was measured by flow cytometry (FCM; undetectable MRD <10-4) in peripheral blood (PB) and circulating tumor DNA (ctDNA) using digital droplet polymerase chain reaction of variable-diversity-joining (VDJ) rearrangements and CLL-related mutations in plasma. The median number of previous treatments was 1 (range, 1-4); 18 patients (40%) had received a Bruton tyrosine kinase inhibitor (BTKi) and/or venetoclax before inclusion, 14 of 44 (31.8%) had TP53 aberrations, and 34 (75.6%) had unmutated immunoglobulin heavy-chain variable region genes. With a median observation time of 36.3 months and all patients off-treatment for a median of 21.9 months, uMRD <10-4 in PB was achieved in 42 of the 45 patients (93.3%) at any time point, including 17 of 18 (94.4%) previously exposed to venetoclax/BTKi and 13 of 14 (92.9%) with TP53 aberrations. The estimated 3-year progression-free and overall survival rates were 85.0% and 93.8%, respectively. Overall, 585 paired FCM/ctDNA samples were analyzed and 18 MRD recurrences (5 with and 13 without clinical progression) occurred after the end of treatment. Twelve samples were first detected by ctDNA, 3 by FCM, and 3 synchronously. In conclusion, time-limited MRD-guided acalabrutinib, venetoclax, and obinutuzumab achieved deep remissions in almost all patients with relapsed/refractory CLL. The addition of ctDNA-based analyses to FCM MRD assessment seems to improve early detection of relapses. This trial was registered at www.clinicaltrials.gov as #NCT03787264.