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Targeting Minimal Residual Disease Emerges as a Viable Path Toward Cancer Cure

This Nature Reviews Cancer perspective examines how new functional approaches can characterize clonal heterogeneity and predict therapeutic sensitivity of minimal residual disease at the single-cell level. Despite dramatic responses to targeted therapies including kinase inhibitors and immune checkpoint blockers, most patients retain MRD that drives relapse. The authors present preliminary evidence that iterative detection, profiling, and targeting of MRD could meaningfully improve outcomes and potentially achieve cure.

The original study

Targeting minimal residual disease: a path to cure?

Authors
Luskin MR, Murakami MA, Manalis SR, Weinstock DM
Journal
Nature reviews. Cancer
Type
Journal Article, Research Support, N.I.H., Extramural, Research Support, Non-U.S. Gov't, Review
PMID
29376520
Read the original study →

Original abstract

Therapeutics that block kinases, transcriptional modifiers, immune checkpoints and other biological vulnerabilities are transforming cancer treatment. As a result, many patients achieve dramatic responses, including complete radiographical or pathological remission, yet retain minimal residual disease (MRD), which results in relapse. New functional approaches can characterize clonal heterogeneity and predict therapeutic sensitivity of MRD at a single-cell level. Preliminary evidence suggests that iterative detection, profiling and targeting of MRD would meaningfully improve outcomes and may even lead to cure.